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HP HP0-S45 : Delta - Integrating HP ProLiant Server Solutions Exam

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Test Number : HP0-S45
Test Name : Delta - Integrating HP ProLiant Server Solutions
Vendor Name : HP
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HP0-S45 exam Format | HP0-S45 Course Contents | HP0-S45 Course Outline | HP0-S45 exam Syllabus | HP0-S45 exam Objectives


Exam ID : HP0-S45
Exam type : Proctored
Exam duration : 2 hours
Exam length : 60 questions
Passing score : 68%
Delivery languages : English, Japanese

This exam has 60 questions. Here are types of questions to expect:
- Matching
- Multiple choice (multiple responses)
- Multiple choice (single response)
- Drag-and-drop
- Point and click

The exam tests your implementation knowledge and skills on HPE ProLiant server products and solutions. syllabus covered in this exam include server architectures and associated technologies and their functions, features, and benefits. Additional syllabus include how to plan preinstallation activities; install and configure; tune and optimize; troubleshoot and repair; monitor, administer, and operate HPE server solutions.

Candidates who want to acquire the new HP ASE - ProLiant Server Solutions Integrator V2 certification and have already acquired the previous version - HP ASE - Server Solutions Integrator V8.1. Although anyone may take the exam, they recommend that candidates have a minimum of two years implementation experience with HPE ProLiant server solutions. Candidates are expected to have industry standard technology knowledge from training or hands on experience.

Describe, explain, and differentiate foundational server architectures and technologies
- Describe and differentiate server processor technologies
- Describe and differentiate server memory technologies
- Describe and differentiate server storage technologies
- Describe and differentiate server networking technologies
- Explain high availability concepts
- Describe and differentiate server power technologies
- Explain disaster recovery concepts(battery backup, HPE Smart Storage Battery, remote key management)
- Describe virtualization solutions
- Identify current server OS
Describe and differentiate the functions, features, and benefits of HPE ProLiant server products and solutions
- Describe ProLiant DL servers and associated products
- Describe ProLiant BL servers and associated products
- Describe HPE datacenter and rack infrastructure solutions
- Differentiate HPE legacy BIOS vs. UEFI
- Describe HPE Administrative Tools(health and fault technologies; e.g. HP OneView)
- Describe HPE SmartArray products
- Describe HPE Flexfabric products
Explain pre-installation planning activitiesfor HPE server solutions
- Explain hardware prerequisite requirements
- Explain software prerequisite requirements
- Determine optionalconfiguration setups based on newly discovered requirements
Describe how to install, configure, set up, validate, and document HPE server solutions
- Determine appropriate power requirements
- Describe how to install rack and accessories
- Describe how to meet servercooling requirements
- Describe how to install and configure server hardware, infrastructure, and expansion options(processors, memory, internal storage, networking, etc.)
- Describe how to install and configure HPE external storage
- Describe how to configure server and related options
- Describe how to install an HPE supported operating system
- Describe how to install and configure HPE management software (HP OneView)
- Describe how to validate the operating environment of the new installation using HP OneView, event logs, diagnostictools, etc.
Describe how to tune, optimize, and upgrade HPE server solutions
- Describe the performance management process
- Determine appropriate HP management toolsto tune, optimize, and upgrade HPE server solutions
Describe how to troubleshoot, repair, and replace HPE server solutions during installation
- Describe when and how to use the HPE 6-Step Troubleshooting Methodology during installation and in established HPE environments
- Describe how to implement preventive measures and prevent issue reoccurrence
Describe how to manage, monitor, administer, and operate HPE server solutions
- Describe how to use HPE Server Management Tools(OA, Insight Display, HP OneView)
- Describe how to manage and maintain the operating system
- Describe how to implement service essentials(Insight Remote Support)



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HP Delta exam Braindumps

Gene therapy in a affected person with Sickle mobile ailment | HP0-S45 exam braindumps and Questions and Answers

Engraftment and Gene Expression figure 1. figure 1. Engraftment with Transduced Cells and Therapeutic Gene Expression within the patient.

Panel A shows vector copy number values in blood nucleated cells and the brief-lived CD15+ (neutrophils) fraction thereof over 15 months after infusion of transduced CD34+ cells. preliminary values in transduced cells earlier than the infusion are proven. Panel B suggests total hemoglobin levels and calculated tiers of each and every hemoglobin fraction based on high-efficiency liquid chromatography measurements of globin chains. The p.c contribution of hemoglobin fractions at month 15 is additionally indicated. The hemoglobin A (HbA) ranges are derived from the commonplace red-cell transfusions got by means of the affected person earlier than gene remedy and in brief thereafter (the last pink-phone transfusion came about on day 88). HbA2 is an choice grownup hemoglobin that is not derived from transfused blood. HbF denotes fetal hemoglobin, and HbS sickle hemoglobin.

Neutrophil engraftment became performed on day 38 after transplantation, and platelet engraftment turned into finished on day ninety one after transplantation. determine 1A shows the trajectory of vector copy numbers and figure 1B shows production of HbAT87Q. Gene marking increased steadily in complete blood, CD15 cells, B cells, and monocytes (Fig. S2 within the Supplementary Appendix), stabilizing three months after transplantation. increases in stages of vector-bearing T cells had been more gradual.

HbAT87Q degrees additionally expanded incessantly (determine 1B) and purple-mobile transfusions have been discontinued, with the last transfusion on day 88. levels of HbAT87Q reached 5.5 g per deciliter (46%) at month 9 and persisted to raise to 5.7 g per deciliter (48%) at month 15, with a reciprocal lower in HbS stages to five.5 g per deciliter (forty six%) at month 9 and 5.eight g per deciliter (49%) at month 15. total hemoglobin ranges had been sturdy between 10.6 and 12.0 g per deciliter after post-transplantation month 6. Fetal hemoglobin degrees remained under 1.0 g per deciliter.

safety

The affected person had anticipated facet effects from busulfan conditioning. Grade 3 and 4 hobbies blanketed grade four neutropenia, grade three anemia, grade 3 thrombocytopenia, and grade 3 an infection with Staphylococcus epidermidis (with superb consequences on blood tradition), all of which resolved with ordinary measures. After the affected person turned into discharged from the sanatorium, four grade 2 opposed routine have been reported: decrease limb ache three months after medicine and transient raises in alanine aminotransferase, aspartate aminotransferase, and γ-glutamyltransferase between 5 and 8 months after medicine. All these hobbies resolved spontaneously.

No adversarial activities concerning the LentiGlobin BB305–transduced stem cells had been suggested (table S1 within the Supplementary Appendix). test consequences for the presence of replication-able lentivirus were uniformly negative. Serial monitoring of integration websites in peripheral-blood samples confirmed a constantly polyclonal profile devoid of detection of a dominant clone (described as a single clone accounting for >30% of wonderful integration movements) through month 12 (Fig. S3 in the Supplementary Appendix).

medical and Biologic Measures

The patient was discharged on day 50. greater than 15 months after transplantation, no sickle cell ailment–connected medical pursuits or hospitalization had passed off; this contrasts favorably with the duration earlier than the patient began to get hold of regular transfusions. All medicinal drugs had been discontinued, together with pain medication. The patient said full participation in average academic and physical activities. Magnetic resonance imaging (MRI) of the top at 8 months showed unchanged punctate subcortical white-matter hypodensities. decrease limb MRI at 14 months showed no fresh bone or tissue damage.

table 1. desk 1. Key Laboratory Values earlier than Gene therapy (at Screening) and at 3-Month Intervals after Infusion of Transduced CD34+ Cells.

alterations in sickle telephone disease–related biologic measures are shown in table 1. comprehensive blood counts had been sturdy, reticulocyte counts lowered substantially (Fig. S4 within the Supplementary Appendix), and circulating erythroblasts had been now not detected. Laboratory values, together with urinary microalbumin tiers, indicated standard renal and liver functions. besides the fact that children iron chelation turned into discontinued before transplantation, the ferritin ranges lowered to 363 μg per liter at month 15, and MRI of the liver 1 12 months after medicine showed a low iron load (level of mobilizable circulating iron, relaxation expense R2*=117 Hz; and iron level, 3.1 mg per gram vs. fifty four Hz and 14.6 mg per gram before gene therapy). Plasma stages of complete bilirubin and lactate dehydrogenase normalized. Soluble transferrin receptor levels superior and had been three.four instances as high as average values at screening and 1.5 instances as high at months 12 and 15, indicating modern normalization of erythropoiesis.

determine 2. determine 2. outcomes of Sickle cell disorder–selected crimson-telephone Assays.

Panel A shows prices of crimson-mobilephone sickling beneath normoxic conditions (20% oxygen saturation) and Panel B indicates fees of pink-telephone sickling beneath hypoxic circumstances (10% oxygen saturation) within the patient at 6 months and one year after gene remedy and amongst control sufferers from whom purple-telephone samples had been bought: two patients with heterozygous A/S “sickle trait” (Controls 1 and 2; handle 1 is the affected person’s mother) and three patients with sickle phone disease (Controls three, four, and 5). equivalent consequences had been got at 7% and 5% oxygen saturation charges (statistics no longer proven). T bars indicate typical errors. Panel C shows oxygen dissociation curves for red cells 12 months after gene therapy within the patient and in the patient’s heterozygous (A/S) mom (control 1). These analyses have been carried out concurrently, under identical situations. The imply red-phone deoxygenation curve (strong black line) and the imply crimson-telephone reoxygenation curve (dashed black line) for 15 untreated patients with sickle telephone sickness are additionally shown. Panel D suggests purple-cell deformability 365 days after gene therapy in the patient as in comparison with his heterozygous (A/S) mom (manage 1) and an extra patient with sickle phone disease (control 6). The grey zone demarcates the latitude inside which 95% of non–sickle phone sickness crimson cells fall, and the black curve is the mean curve for in shape individuals. The elongation index changed into calculated because the ratio of the length (A) and width (B) of a cellphone, the place (A−B) was divided through (A+B), and the outcome was expressed as a decimal between 0 and 1. Panel E shows the purple-telephone density profile 365 days after gene therapy within the affected person, obtained with using a phthalate gradient. They measured 10 samples (indicated with the numbers 1 through 10 on the black curve) at a number of phthalate densities. purple traces demarcate three distinct densities of cells: low (<1.086 mg per milliliter), medium (1.086 to 1.096 mg per milliliter), and high (>1.096 mg per milliliter). Orange lines point out limits of a standard profile. The values for the affected person are shifted to the left on account of the linked single α-globin gene deletion. Cells denser than 1.110 mg per milliliter of phthalate answer are considered to be dense cells.

since the affected person acquired a daily transfusion routine for four years before this analyze and because of the change transfusion before transplantation, significant comparative reviews before and after transplantation could not be conducted. although, the proportions of sickled purple cells in the patient’s blood at months 6 and 12 had been tremendously lower than these in untreated patients with sickle mobile disorder (βS/βS) (figure 2A). At month 12, the sickling cost in hypoxic situations become not enormously distinct from that of the affected person’s asymptomatic, heterozygous (βA/βS) mother (determine 2B). Oxygen dissociation experiences, which quantify oxygen saturation relative to the partial drive of oxygen, confirmed that consequences within the affected person at month 12 and consequences in a heterozygous (βA/βS) handle had been similar (determine 2C and 2d).


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